The Spike Protein, Amyloids, Cyanide and the Inhibition of Complex IV of the Electron Transport Chain
May 1, 2022
What we may be dealing with is an accidentally released “work in progress.” This “work in progress” may be an attempt to make a transmissible “Zyklon-B” bioweapon.
After continuing analysis and examination of the Spike Protein and its interactions with the mitochondria, I have made a startling discovery.
In June of 2020, a paper was published which hypothesized COVID-19 could be completely induced by viral interference with the mitochondria alone.
https://journals.physiology.org/doi/full/10.1152/ajpcell.00224.2020
It has also been discovered that the virus inhibits Electron Transport Chain Complex IV. In particular, interactions of viral proteins with other components of complex I and complex IV have been observed, which could lead to imbalanced RC complex activity and ROS production.
https://www.ncbi.nlm.nih.gov/pmc/articles/PMC8473126/
No other virus has this strong of an effect on the mitochondria. This has been proven by an RNA-GPS analysis predicting where the SARS-CoV-2 genome localizes to. The Spike Protein has a VERY strong affinity for the Mitochondrial Matrix. It is from here that it can inhibit the functionality of Complex IV.
So, what is startling beyond the already described pathology?
Zyklon-B was used by the Germans to conduct mass genocide in World War II. Zyklon-B is a Hydrogen Cyanide poison.
How does Cyanide kill?
CYANIDE INHIBITS COMPLEX IV OF THE ELECTRON TRANSPORT CHAIN.
This also fits the Amyloidosis Hypothesis for COVID-19 and Long COVID.
IN THE EXACT SAME MANNER OF INHIBITION. AMYLOID-B INHIBITS COMPLEX IV OF THE ELECTRON TRANSPORT CHAIN.
Because acute cyanide poisoning may impact multiple organ systems in addition the highly oxygen sensitive central nervous and cardiovascular systems, initial clinical signs may be nonspecific and vague such as headache, nausea, vomiting, anxiety, agitation, and confusion. With severe poisonings, lethargy, seizures and coma are potential manifestations of CNS toxicity. While terminal findings of hypotension and bradycardia, resulting in cyanosis are typical of severe cardiovascular toxicity, clinicians may briefly encounter tachycardia and hypertension due to CATECHOLAMINE MEDIATED reflex compensatory processes prior to hemodynamic embarrassment.
One can certainly and easily make the case that the Spike Protein is an aerosolized, highly transmissible “Zyklon-B.”