Recent data have indicated that a catastrophic loss of telomeric sequences might, in some cases, precede the apparent onset of DNA damage-induced apoptosis in the absence of both telomerase and the check-point regulator p53.

S2 RNA lowers telomerase and the spike also reduces p53,  causing the perfect storm for massive telomere deletion.

This removes the "armor" protecting our DNA allowing for almost certain oncogenesis. A comparison would be removing your skin. A fatal infection would be guaranteed to occur.

Most cells renew and proliferate, meaning  that over time the telomeres would be intact in new cells. Our heart cells and neurons do not renew, so once stripped of their telomeres, they are fair prey for oncogenesis. Even the cells which renew would be laid bare for a period of time and open to the same possible genetic manipulation.

To make matters even worse, and, I believe, to prove the bioweapon aspect of the Spike Protein, damaged telomeres INCREASE the expression of ACE2, leading to more viral invasion, replication, p53 suppression and telomere deletion. This induces a fatal  feedback loop.

I do now know how eukaryotic organisms survive this, if it has the same effect on other animals. This may explain China's draconian actions. What do we do, in the long run? Even if the spike protein therapies are stopped, as they must be. The spike causes this.

Referenced/Related Papers

SARS-CoV-2 Spike and Telomerase RNAs Compared to Arrive at an Explanation for Increased Ageing in Alveolar Cells in Severe COVID-19

https://www.preprints.org/manuscript/202103.0551/v1

SARS-CoV-2 Mediated Endothelial Dysfunction: The Potential Role of Chronic Oxidative Stress

https://www.frontiersin.org/articles/10.3389/fphys.2020.605908/full

Clues to catastrophic telomere loss in mammals from yeast telomere rapid deletion

https://pubmed.ncbi.nlm.nih.gov/14634639/

DNA damage response at telomeres boosts the transcription of SARS-CoV-2 receptor ACE2 during agin

https://www.biorxiv.org/content/10.1101/2021.06.09.447484v1.full

S2 subunit of SARS-nCoV-2 interacts with tumor suppressor protein p53 and BRCA: an in silico study

https://www.sciencedirect.com/science/article/pii/S1936523320303065