August 22, 2021
EPIGENITIC MEMORY: THE SPIKE PROTEIN'S PROGRAMMING OF (NOT FULLY) SENESCENT CELLS. THAT REPLICATE AS SENESCENT CELLS. A “ZOMBIFIED" LIFE SENTENCE. COULD ANYTHING BE THIS CRUEL? “THE FLY" VIRUS?
In the paper by Gaetano, et al., EVIDENCE FOR
BIOLOGICAL AGE ACCELERATION AND TEELOMERE SHORTENING IN COVID19 SURVIVORS, the conclusion arrives at the following statement:
“However, a warning might be raised that sequelae of SARS-CoV-2 infection might rely on persistent epigenomic modifications, possibly underlying the presence of a COVID19 epigenetic
What is “epigenetic memory?" Epigenetic memory is an essential process of life that governs the inheritance of predestined functional characteristics of normal cells and the newly acquired properties of cells affected by cancer and other diseases from parental to progeny cells.
As I stated, what if the senescence was a life sentence? Could anything be this cruel? Could the Spike Protein program cells to be senescent – but not fully senescent? Senescent cells can have senescent progeny.
The Ability to Generate Senescent Progeny as a Mechanism Underlying Breast Cancer Cell Heterogeneity. Yes, you read that correctly. Senescent cells are able to generate senescent progeny. An “unending" chain of defective cells, replacing previously healthy, fully functioning cells.
More importantly, when the senescent progeny were examined, none of the clones tested over a long period of time (>60 PDs) entered full senescence. This is akin to the final scene of The Fly, when Jeff Goldblum's character, fully transformed into a non-functioning, genetically destroyed, barely alive “fly," pathetically begs Geena Davis' character to euthanize him.
I have read so many emails and heart wrenching accounts from and of Long COVID sufferers, that their suffering invokes those feelings in me.
The Spike Protein is thought to have epigenetic implications: The N501Y variant, in particular, has been suggested to be 56% more transmissible, owing to the enhanced binding affinity of the Spike protein to its polypeptide receptor, ACE2, on the host cell; a finding with potential epigenetic implications.
Senescent cells can be removed, of course. But does the virus remain? Lurking, latent, to reactivate later and induce the nightmare all over again? We don't know. I certainly hope not.
The darkest side? A genetic setup where you literally have to purchase “anti-viral anti-malware" to keep your system clean.
It would seem best to stop the epigenetic modifications from being made in the first place. Ketones, and compoinds like β-hydroxybutyrate, prevent your DNA from being accessed. A true “Access Denied." Perhaps this is why those who have low blood sugar and those who fast, seem to fare much better against the virus. But are the changes still happening, at a slower pace?
Is the denial of NAC and the dismissal of Niacin and IVY, to prevent “unofficial software" from being employed? We have been instructed our genes are now an “operating system."
Are we to live as those in Dune? Instead of a heart plug, threat of a life sentence of Long COVID for non-compliance? I hope, as other scientists have done, this will be my one step too far.
The Ability to Generate Senescent Progeny as a Mechanism Underlying Breast Cancer Cell Heterogeneity
Methylation Pathways and SARS-CoV-2 Lung Infiltration and Cell Membrane-Virus Fusion Are Both Subject to Epigenetics
Epigenetic memory in development and disease: unraveling the mechanism
EVIDENCE FOR BIOLOGICAL AGE ACCELERATION AND TELOMERE
2 SHORTENING IN COVID19 SURVIVORS