February 21, 2021
It is very likely that the Spike Protein is using the same Voltage-dependent calcium channel signaling to induce dendritic cell migration to the CNS and other organs. 30% of humanity is infected with latent Toxoplasma Gondii. Could SARS-CoV-2 be activating T. Gondii? Could it be MIMICKING T. GONDII? Please note symptoms of Toxoplasmosis:
Swollen lymph nodes
Reactivation of chronic T. gondii infection can cause life-threatening TE in immunocompromised individuals. Here, we show for the first time that T. gondii infection in CNS leads to astrocyte polarization to A1 subtype, which potentially harms neurons.
A1 astrocytes have been reported in the lipopolysaccharide (LPS)-induced CNS inflammation model, traumatic brain injury (TBI), and prion diseases.
Voltage-dependent calcium channel signaling mediates GABAA receptor-induced migratory activation of dendritic cells infected by Toxoplasma gondii
Prion Disease, Toxoplasmosis, and Vasculitis