Spike Protein Invades Neurons Like Toxoplasma Gondii
February 21, 2021

It is very likely that the Spike Protein is using the same Voltage-dependent calcium channel signaling to induce dendritic cell migration to the CNS and other organs. 30% of humanity is infected with latent Toxoplasma Gondii. Could SARS-CoV-2 be activating T. Gondii? Could it be MIMICKING T. GONDII? Please note symptoms of Toxoplasmosis:


Body aches

Swollen lymph nodes

Headache

Fever

Fatigue

Headache

Confusion

Poor coordination

Seizures

Lung Problems


Reactivation of chronic T. gondii infection can cause life-threatening TE in immunocompromised individuals. Here, we show for the first time that T. gondii infection in CNS leads to astrocyte polarization to A1 subtype, which potentially harms neurons.


A1 astrocytes have been reported in the lipopolysaccharide (LPS)-induced CNS inflammation model, traumatic brain injury (TBI), and prion diseases.

Referenced/Related Papers

Voltage-dependent calcium channel signaling mediates GABAA receptor-induced migratory activation of dendritic cells infected by Toxoplasma gondii

https://journals.plos.org/plospathogens/article?id=10.1371/journal.ppat.1006739


Prion Disease, Toxoplasmosis, and Vasculitis

https://18kxerroa80uqzne1b7qmoje-wpengine.netdna-ssl.com/wp-content/uploads/2018/06/NM-Brain-Lecture-013-Prions-Toxoplasmosis-and-Vasculitis.pdf