March 5, 2022
THE ENTIRE DISEASE (AND LONG COVID) IS CAUSED BY THE SPIKE PROTEIN DAMAGING THE ENDOTHELIUM AND IMPAIRING FIBRINOLYSIS RESULTING IN MICROCLOTTING.
There is little question that clotting, in some form, accompanies severe Covid-19 pneumonia. The D-dimer level in such patients is routinely elevated—yet gross clots are detected in only a minority. That accounts for the D-dimer elevation in the remaining patients? And, does that elevation truly reflect the presence of widespread intravascular clotting? If that is the case, then the pathophysiology of severe pneumonia is primarily that of a clotting disorder in many, perhaps most, ICU Covid-19 patients.
It IS NOT, nor has it ever been “Pneumonia!”
This FINALLY explains why it appears to be a high altitude disease! O2 exchange impairment!
In fact, a paper from a year ago (March 2021) showed that SARS-CoV-2 spike protein S1 induces fibrin(ogen) resistant to fibrinolysis. This allows for the microclotting which is INVISIBLE by normal testing methods. It would also have been invisible during Spike Protein therapy trials.
Not only this, but the induced autoimmune disease being observed is also explained by these findings as extensive fibrin amyloid microclots that can persist, can entrap other proteins, and that may lead to the production of various autoantibodies.
THE MYOCARDITIS BEING OBSERVED IS FROM THE SPIKE PROTEIN INDUCING THIS VERY MICROANGIOPATHY! A Case Report: Co-occurrence of Myocarditis and Thrombotic Microangiopathy Limited to the Heart in a COVID-19 Patient demonstrates this mechanism.
Clearly, this also explains the clotting issues observed after the application of Spike Protein therapies.
THE “BIZARRE” IMAGES BEING SEEN IN BLOOD POST THERAPY MAY BE THESE MICROCLOTS!
Referenced/Related Papers
https://www.preprints.org/manuscript/202202.0347/v1
SARS-CoV-2 spike protein S1 induces fibrin(ogen) resistant to fibrinolysis: implications for microclot formation in COVID-19
https://www.semanticscholar.org/paper/SARS-CoV-2-spike-protein-S1-induces-fibrin(ogen)-to-Grobbelaar-Venter/f106f2f61d08caa40e221ba8452f299e7e9ec1e9
A central role for amyloid fibrin microclots in long COVID/PASC: origins and therapeutic implications https://portlandpress.com/biochemj/article/479/4/537/230829/A-central-role-for-amyloid-fibrin-microclots-in
Studies look at clotting, myocarditis tied to COVID-19 vaccines
https://www.cidrap.umn.edu/news-perspective/2021/08/studies-look-clotting-myocarditis-tied-covid-19-vaccines
Case Report: Co-occurrence of Myocarditis and Thrombotic Microangiopathy Limited to the Heart in a COVID-19 Patient
https://www.frontiersin.org/articles/10.3389/fcvm.2021.695010/full