The Spike Protein and Dectin-2 family of C-type lectins (explaining the appearance of Mucormycosis and Autommune Disease in relation to COVID-19 and Spike Protein therapies)
May 22, 2021
Given the recent finding that ~800 billion spikes are cirulating in the blood after each mRNA injection, one looks to other receptors for the spike protein.
One such receptor is the family of C-type lectins. These are a diverse group that contain Dectin-2. These are located in the TELOMERIC REGION of the NK-gene cluster. The Dectin-2 lectins assist the body in pathogen recognition including fungi, viruses and probably others. Many also recognize endogenous ligands, highlighting the importance of this family in homeostasis. Indeed, the Dectin-2 locus has been identified as a susceptibility region associated with autoimmune disorders, including multiple sclerosis, systemic lupus erythematosus, rheumatoid arthritis and type I diabetes.
It is most likely the case that the free floating spike proteins are toxifying C-type lectins, including Dectin-2 causing either a depletion via eipgentics or autoantibodies.
The body is being set up for severe acute infections, cancer and autoimmune disease.
Referenced/Related Papers
SARS-CoV-2 exacerbates proinflammatory responses in myeloid cells through C-type lectin receptors and Tweety family member 2
https://cell.com/immunity/fulltext/S1074-7613(21)00212-0
C-Type Lectin-Like Receptors As Emerging Orchestrators of Sterile Inflammation Represent Potential Therapeutic Targets
https://frontiersin.org/articles/10.3389/fimmu.2018.00227/full
The Dectin-2 family of C-type lectins in immunity and homeostasis