THE SPIKE PROTEIN OF SARS-CoV-2 IS A CUE BALL THAT DYNAMICALLY ACTIVATES HUMAN ENDOGENOUS RETROVIRUSES. SARS-CoV-2 DOESN'T KILL YOU. SARS CoV-2 CAUSES YOU TO KILL YOU.
If you look at Pulmonary Arterial Hypertension, you notice all the hallmarks of those who die from SARS-CoV-2 infection. Indeed, those who survive COVID-19 appear to be predisposed to develop this fatal illness.
Pulmonary arterial hypertension (PAH) is a deleterious disease of the lung vasculature characterized by endothelial dysfunction, medial hypertrophy and proliferation, and eventual occlusion of the pulmonary arterioles. Particularly note that altered inflammatory dynamics, such as hematopoietic and myeloid expansion, B- and T-cell dysfunction, increased cytokine production, circulating autoantibodies, and tertiary lymphoid tissue neogenesis, may initiate or drive PAH.
Is this not COVID-19? And, the cause of PAH dystregulation has been elusive. Activation of Human Endogenous Retrovirus K is most likely the cause of PAH.
SCHIZOPHRENIC PATIENTS HAVE HERV-W ENV ANTIBODIES
In schizophrenic patients, positive antigenemia for ENV was found in 23 of 49 (47%) and for GAG in 24 of 49 (49%). Only 1 of 30 (3%) for ENV and 2 of 49 (4%) for GAG were positive in blood donors (p < .01 for ENV; p < .001 for GAG). Interestingly, bioclinical data analyses revealed significant correlation between GAG or ENV antigenemia (a protein causing dysimmune inflammatory effects) and C-reactive protein (CRP) levels (a systemic inflammation biomarker). Frequently elevated CRP has previously been described in schizophrenic patients and has been shown to match with an evolution toward cognitive deficit and neuronal loss.
This is the "Ghost in the Machine."
Please note that peptides in the Spike Protein is SARS-CoV-1 was found to be HOMOLOGOUS with our HERVs (Syncytin). This was discussed in a paper from WUHAN back in 2005.
Structural characterization of the fusion core in syncytin, envelope protein of human endogenous retrovirus family W
COVID-19 and Pulmonary Arterial Hypertension: Early Data and Many Questions
COVID-19 patients may become predisposed to pulmonary arterial hypertension
Antibody against envelope protein from human endogenous retrovirus activates neutrophils in systemic lupus erythematosus