LETHAL RECIPROCATION: THE IMMUNE RESPONSE TO THE SPIKE PROTEIN VS. THE SPIKE PROTEIN’S INACTIVATION OF TELOMERASE – AN EXPLANATION FOR THE RECENT EVENTS IN ISRAEL
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August 14, 2021

The "robust immune response" generated by the spike protein may come at a lethal cost. A recent paper published in the Journal of Bacteriology & Parasitology convincingly demonstrates that the Spike Protein has deleterious effects on telomerase, impairing its synthesis. With this activity is inhibited, it takes time for the inevitable immune cell exhaustion to appear. This may be the signature Trojan Horse "gift" of the spike. Whatever short-lived immune response is fool's gold replacing the ability of the immune system to replenish itself. This explains what we are currently observing in Israel.

 

In fact, all of the long term effects of SARS-CoV-2 infection suggest that the spike protein may be chronically present in some, continually suppressing telomerase. 

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In a fabulous lecture (referenced) Elizabeth Blackburn demonstrates that without telomerase, key cells in the body are unable to replenish themselves and "run out" well before they should in the course of a normal lifespan. 


We are basically seeing the effects of CHEMOTHERAPY with the spike protein. But in a very unique way. Quickly replicating cells are being exhausted.


We can now explain the all too common reports of hair  loss, for example. 


It does appear that telomerase activity returns once the virus is cleared, and may be ameliorated in the case of chronic infection.


The Spike Protein cannot be used for immunogenicity. The momentary strong immune response is a false hurrah. Stop this now. We who are about to die, salute the Spike Protein.


Repeated injections will almost certainly be lethal.

Referenced/Related Media



Sars-Cov2 Spike and Telomerase RNA’s Compared to Arrive at an Explanation for Increased Ageing in Alveolar Cells in Severe COVID-19

https://longdom.org/open-access/sarscov2-spike-and-telomerase-rnas-compared-to-arrive-at-an-explanation-for-increased-ageing-in-alveolar-cells-in-severe.pdf