A paper from 2006 revealed a very interesting (and for us, now, fortuitous) phenomenon. If the prion protein is overexpressed, it chelates copper. It is known that the Spike Protein of SARS-CoV-2 mimics the human prion protein. It is highly likely that the body perceives this as an overexpression of the prion protein. 

Overexpression of PrPC induces strong alterations at different steps of T cell maturation. On TgA20 mice, we observed that these alterations are cell autonomous and lead to a decrease of ?? T cells and a concomitant increase of ?? T cell numbers. PrPC has been shown to bind and chelate copper and, interestingly, under a copper supplementation diet, 

But, there is another, very rare disease that occurs with low serum copper and high serum ferritin - the EXACT conditions we see in COVID.

Aceruloplasminemia

This disease is very difficult to identify. In fact, it took OVER 20 YEARS in one patient to identify it! And, it has all the hallmarks of COVID/Long COVID.

At the time of diagnosis, the man was almost 60 years old. He had significant iron deposition in the brain and showed cognitive impairment, dementia, chronic fatigue and neurological symptoms consistent with cerebellar damage, such as difficulties with coordination and gait, dysarthria, intention tremor and dysdiadochokinesia.

Please read the referenced case study. I believe this offers tremendous hope for solving the pandemic. I also believe it makes absolutely clear we must immediately cease all Spike Protein therapies immediately.