VASCULAR DISEASE AS CAMOUFLAGE: THE SPIKE PROTEIN OF SARS-CoV-2 AS MICROTUMOR
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January 1, 2022

Respiratory disease held the world in a false awe at the beginning of the COVID-19 pandemic. Health authorities the world over reiterated the same narrative: We have a new Respiratory Virus. Those of us who looked closely at the disease from the beginning knew that wasn’t the true picture – or at least not the full picture.


Then, slowly, over the course of the first year of the pandemic, everyone else began to realize this, too. Finally, towards the end of 2020, COVID-19 was universally recognized as a Vascular Disease. However, I have realized the virus’ deception does not stop there.


IT IS ONLY A VASCULAR DISEASE INSOMUCH AS THE SPIKE PROTEIN MUST BREAK THROUGH THE VASCULATURE IN ORDER TO INVADE ORGANS. Precisely. The. Same. Mechanism. Cancer. Employs.


Cancer kills by growing into key organs, nerves, or blood vessels and interfering with and impairing their function. It can begin in almost any human cell. Yet, isn’t this EXACTLY what the Spike Protein is doing? It rips through the endothelium to invade organs and impair their function. The much greater danger is that instead of beginning in “any” cell, the spike is attempting to do this in “every” cell.


Cancer disrupts the cellular destruction and renewal process. As a result, new cells become increasingly abnormal, and old cells live when the body should destroy them. Isn’t this what we are observing?


Let us review the key ways cancer kills and their relation to the Spike Protein.


MALNOURISHMENT


Malnutrition is a major cause of mortality in advanced cancer. This can happen when tumors in the digestive system form a blockage or interfere with the absorption of essential nutrients.


The spike protein does this in a very clever way. It robs you of your electrolytes, which are the result of the body’s processing of nutrients.


SARS-CoV spike proteins can compete for electrolytes in physiological fluids according to structure-based quantum-chemical calculations

https://pubmed.ncbi.nlm.nih.gov/34395179/


LACK OF OXYGEN


Lung cancers especially cause a lack of oxygen, another device which cancer uses to kill  you. Again, the Spike Protein is able to mimic this on its own in a very sophisticated way.


Toward this end, we have recently begun to investigate the interplay between oxygen transport, oxygen sensing, and mitochondrial metabolism in human pulmonary arterial endothelial cells (HPAEC) by exploring, first the impact of COVID-19 spike protein on these parameters in the presence of Hb and under normoxic and hypoxic cell culture conditions. We found that the spike protein alone induced changes in cells without the entry of the virus and that cell-free Hb did not attenuate the effects of the SARS-CoV-2 S1 spike protein in vitro (Jana et al., 2021). We also begun to investigate the three elements of homeostasis (oxygen transport, oxygen sensing, and mitochondrial pathways) in lungs from hamster model of COVID-19 infections (Selvaraj et al., 2021) to verify the interconnectivity of these pathways in living biological systems.


COVID-19 infection clearly impacts oxygen sensing elements as seen in changes with HIF-target genes as well as mitochondrial respiration in response to low oxygen levels in circulation.


The Impact of COVID-19 Infection on Oxygen Homeostasis: A Molecular Perspective

https://www.frontiersin.org/articles/10.3389/fphys.2021.711976/full


LIVER DAMAGE


Liver damage is also one of the key factors in the fatal consequences of Cancer. Indeed, the Spike Protein damages the liver, impairing its function.


Functional characterization of liver from patients with COVID-19. a Immunofluorescence analyses of ALB, CK19, ACE2, and spike proteins expressed in liver tissues from COVID-19 patients and control individuals (scale bar: 50 and 10 μm). Virus proteins (spike) were found in the damaged nucleus.


Pathological features of COVID-19-associated liver injury—a preliminary proteomics report based on clinical samples

https://www.nature.com/articles/s41392-020-00406-1


TOO  MUCH CALCIUM


Almost universally, excess calcium, which cancer causes bones to release, can lead to coma and death. Once again, the Spike Protein is able to mimic this mechanism.


It was found that an upstream calcium-dependent PKC isozyme PKC alpha that modulates the downstream ERK/NF-kappaB pathway through an influx of extracellular Ca2+ is induced by the spike protein of SARS-CoV.


Spike protein of SARS-CoV stimulates cyclooxygenase-2 expression via both calcium-dependent and calcium-independent protein kinase C pathways

https://pubmed.ncbi.nlm.nih.gov/17267381/


There are extensive additional papers elucidating each of the points I have made.


It may be that China is not trying to instill a false panic at all. It may be the case that we are dealing with Aerosolized Cancer. What concerns me is that it may not stop at cancer. The prion domains of the spike ring Aerosolized Prion Disease bells.