IS INFILTRATION OF THE SARS-CoV-2 SPIKE PROTEIN INTO ORGAN TISSUE THE CAUSE OF LONG COVID? AND DOES IT AWAIT US ALL?
May 25, 2022
It has been established that the Spike Protein of SARS-CoV-2 induces amyloidosis and fibrils. Yet, is the appearance of amyloids just one of the pathologies it can induce?
Let us consider the findings of fibrin blood clots in patients reporting to the emergency room and in the bodies of the deceased as discovered by embalmers:
Amyloid Beta INDUCES THE FORMATION OF ABNORMAL, DEGRADATION-RESISTANT BLOOD CLOTS. This is paramount in Alzheimer’s Disease (AD).
The association between Aβ and fibrinogen causes altered fibrin clotting, and this aberrant hemostasis could lead to compromised blood flow and increased inflammation, thereby contributing to cognitive decline in AD.
VARIATIONS ON A THEME
Of course, one could argue that Alzheimer’s itself is a VASCULAR DISEASE. This very same mechanism – infiltration of blood vessels with an amyloid protein and eventually the organs themselves – can be induced in every organ. It appears that this is occurring.
It makes sense that the endothelium would be the canary in this coalmine. It is the largest organ in the body and the “road” to all other organs.
What is most fascinating is that results from a study indicated that exogenous Aβ seeds from different Aβ pathologies induced Aβ deposition in the blood vessels rather than the brain parenchyma without being influenced by Aβ strain-specific information, which might be why CAA is a predominant feature of Aβ pathology in iatrogenic transmission cases.
Is that not, by definition, what the Spike Protein is? An Aβ seed from a different Aβ pathology? And is it not inducing “Aβ” deposition in the blood vessels?
In a study of Long COVID patients, the following was determined: There was evidence of mild organ impairment in the heart (32% of patients), lungs (33%), kidneys (12%), liver (10%), pancreas (17%), and spleen (6%). And, surprisingly: “Causality of the relationship between organ impairment and infection cannot be deduced, but may be addressed by longitudinal follow-up of individuals with organ impairment,” the authors said.
However, they are certainly wrong. We absolutely can deduce the causal relationship between organ impairment and infection: it is the amyloid infiltration of the Spike Protein into those organs.
THE DEAD REVEAL HOW THE LION STALKS ITS PREY
If we review autopsies of those who died from COVID-19, there was a finding that gave pause to the researchers. The authors mention the finding casually, yet they have no idea how significant it actually is.
“This post-mortem study showed several histopathological abnormalities in COVID-19 non-survivors; however, none of the findings was specific for direct viral injury, even though SARS-CoV-2 was detected in all examined organs using RT-PCR.”
And this is the point. Those who are not immediately killed by SARS-CoV-2 have, literally, the seeds of destruction sowed in their bodies.
For those who have been following my work, this fits precisely with the accelerated aging I (and others) observed related to SARS-CoV-2 infection.
So, we come full circle. Please read this sentence and contemplate it well:
It is known that hypertension, atherosclerosis and amyloidosis can be viewed as accelerated aging.
Questions to be addressed:
Are the Spike Protein “amyloid seeds” distributed throughout the organs of the infected (and transfected) lying in wait, to be activated by “The heart-ache and the thousand natural shocks that flesh is heir to?” Greatly accelerating any affliction that we experience?
Does the presence of these Spike Proteins mean that Inflammaging is the result of SARS-CoV-2 infection, due to their ability, as a superantigen, to create excessive cytokines?
Are these effects cumulative? With each exposure to the Spike Protein?
Can the Spike Protein be eradicated from the body?
Can the damage done be ameliorated, or reversed?
There is much still to learn.