May 31, 2021
THE CURRENT TREATMENT PROTOCOLS ARE INCORRECT. SARS-CoV-2 IS AN AUTOIMMUNE DISEASE INDUCED BY MITOCHONDRIAL DAMAGE INITIATED BY THE SPIKE PROTEIN. IT MOST CLOSELY RESEMBLES PERIODIC FEVER SYNDROMES: FROM AIDS TO AIDs.
In 1999 Kastner et al first introduced “autoinflammation” describing two diseases characterized by recurrent episodes of SYSTEMIC INFLAMMATION WITHOUT ANY IDENTIFIABLE INFECTIOUS TRIGGER: Familial Mediterranean Fever (FMF) and TNF Receptor Associated Periodic Syndrome (TRAPS).
Characteristic febrile episodes may also be accompanied by chills, abdominal pain, headaches, joint pain (arthralgia), chest pain, and muscle pain (myalgia), stiffness and tightness. Abdominal pain may be associated with nausea, vomiting, and diarrhea or constipation. Individuals with TRAPS may also develop painful inflammation of the delicate membranes that line the inside of the eyelids (conjunctivitis). Additional symptoms that have been reported in males with TRAPS include TESTICULAR PAIN and a higher rate of inguinal hernia than is found in the general population.
A serious complication that occurs in approximately 10-15 percent of TRAPS cases is AMYLOIDOSIS. And so, at long last, we discover why doctors have not been able to find anything causative for Long COVID.
Again. The Spike Protein therapies MUST be stopped. Treatments directed towards autoinflammation6) must be immediately investigated.
Related/Referenced Papers
Likelihood of amyloid formation in COVID-19-induced ARDS
https://www.cell.com/trends/microbiology/fulltext/S0966-842X(21)00067-6
Tumor Necrosis Factor Receptor-Associated Periodic Syndrome
https://rarediseases.org/rare-diseases/tumor-necrosis-factor-receptor-associated-periodic-syndrome/
Mitochondria in autoinflammation: cause, mediator or bystander?
https://www.cell.com/trends/endocrinology-metabolism/pdf/S1043-2760(15)00048-X.pdf
Targeting mitochondria in cancer: current concepts and immunotherapy approaches