Make no mistake. The respiratory effects of SARS-CoV-2 are a dangerous distraction.
Endothelial cells are a nexus of receptors for oncogenesis and neurodegenaration. In addition to having ACE2 receptors, allowing for the invasion of SARS-CoV-2, they also have a7 nAChR receptors. The spike protein antagonizes these receptors and allows for neuroinflammation and neurodegeneration, in addition to interfering with The Cholinergic Anti-inflammatory Pathway. Antagonizing a7 nAChR receptors also DESTABALIZES EPITHELIAL CELL ORGANIZATION. It disrupts epithelium integrity and promotes inflammatory response and tumor development.
The spike protein also alters the metabolism of the body. It causes mitochondrial damage which alters cellular metabolism. The body is put into a state of starvation. It is the same state as cancer cachexia (wasting disease).
SARS IS A HIGHLY SPECIALIZED, BOUTIQUE VIRUS PERFECTLY AND MOST EFFICIENTLY TUNED TO THE EXACT RECEPTORS NEEDED TO PROMOTE CANCER, NEURODEGENERATION AND MULTIPLE SYSTEM ATROPHY (ORGAN FAILURE).
We are dealing with a virus that will eliminate a vast majority of mankind. Putting the spike protein in people allows for this without exposing individuals to the N protein, which is much more likely to put you in the ICU with ARDS.
THERE IS NO IMMUNITY - ONLY ACCELERATION.
Furthermore, a study from Wuhan shows that altering a7 nAChR receptors can have effects on the fetus promoting autoimmune disease.
Role of Cholinergic Receptors in Colorectal Cancer: Potential Therapeutic Implications of Vagus Nerve Stimulation?
α7 nAChR mediated Fas demethylation contributes to prenatal nicotine exposure-induced programmed thymocyte apoptosis in mice
SARS-CoV-2 envelope protein causes acute respiratory distress syndrome (ARDS)-like pathological damage and constitutes an antiviral target